Insertion sequences. No missense mutations, etc. There’s tell tale markers of run-of-the-mill evolutionary mutation, but I’m not a molecular or evolutionary biologist.
So, dude. Reckon sheddase and ACEI, ARB's can be used to counteract the s protein? Or would it make it worse? Clearly there's an imbalance between Angiotensin-2 and Angiotensin-1-7 so surely there's a way to inhibit one or the other, right?
Insertion sequences. No missense mutations, etc. There’s tell tale markers of run-of-the-mill evolutionary mutation, but I’m not a molecular or evolutionary biologist.
What does the cIeavage site do exactly, tho? Help it bind to ACE2? New at this. Learning.
A cleavage site is just that: a location on the enzyme involved with cleaving another protein. ](https://m.youtube.com/watch?v=W1k1sUoLPlA)
Holy crap. Thank you so much. That was so informative.
So, dude. Reckon sheddase and ACEI, ARB's can be used to counteract the s protein? Or would it make it worse? Clearly there's an imbalance between Angiotensin-2 and Angiotensin-1-7 so surely there's a way to inhibit one or the other, right?