The specific mechanism of action of the antiparasitic drug Ivermectin as an antiviral is shown to interfere with the viral replication process by inhibiting nuclear importation of viral proteins by its action against the nucleocapsid protein and RNA dependent RNA polymerase inhibition. Ivermectin has also been shown to reduce cytokine storm by suppressing production of interleukin 6 and tumor necrosis factor alpha - two major components in the cytokine storm. It has also been postulated that Ivermectin can inhibit STAT3 pathway that leads to all kinds of downstream outcomes such as clotting, cytokine storm, hypoxia, etc. Some additional research shows that Ivermectin may bind to ACE2 receptors that can interfere with spike protein binding - but this mechanism of action has been seen by computer studies only and has not been shown in actual in vitro or in vivo studies. More computer modeling studies show that Ivermectin can also bind with CD147 receptors on red blood cells and platelets thereby interfering with the binding of spike protein to these blood components that leads to clotting. Needless to say, there are several different mechanisms where Ivermectin may be useful, not only against viral infection, but also to lessen the effects caused by spike protein binding as a result of injections.
Here is research for those science geeks among us. Repurposing Ivermectin for COVID-19: Molecular Aspects and Therapeutic Possibilities
So, to answer the OP, Ivermectin does not kill the spike protein. Something that is not alive to begin with, cannot be killed. The mechanism of action with Ivermectin is targeted to interfere with viral replication within the cell thereby reducing viral load in an actual infection. Ivermectin may interfere with the spike protein binding on ACE2 and CD147 receptors - but these mechanisms still needs further study. Beyond this, Ivermectin could be useful in mitigating cytokine storm resulting from the pathogenic priming caused by the both types of injections. It may also help combat micro and macro clotting due to spike protein binding.
At the moment, there is nothing that can reverse the production of spike proteins that result from both types of injections for those foolish enough to have received them. All that can be done at this point is to try to lessen the damage and Ivermectin can be used for that purpose. But, Ivermectin does have side effects and should be used with caution as part of a long term strategy to mitigate the effects of injection induced spike protein production.
The specific mechanism of action of the antiparasitic drug Ivermectin as an antiviral is shown to interfere with the viral replication process by inhibiting nuclear importation of viral proteins by its action against the nucleocapsid protein and RNA dependent RNA polymerase inhibition. Ivermectin has also been shown to reduce cytokine storm by suppressing production of interleukin 6 and tumor necrosis factor alpha - two major components in the cytokine storm. It has also been postulated that Ivermectin can inhibit STAT3 pathway that leads to all kinds of downstream outcomes such as clotting, cytokine storm, hypoxia, etc. Some additional research shows that Ivermectin may bind to ACE2 receptors that can interfere with spike protein binding - but this mechanism of action has been seen by computer studies only and has not been shown in actual in vitro or in vivo studies. More computer modeling studies show that Ivermectin can also bind with CD147 receptors on red blood cells and platelets thereby interfering with the binding of spike protein to these blood components that leads to clotting. Needless to say, there are several different mechanisms where Ivermectin may be useful, not only against viral infection, but also to lessen the effects caused by spike protein binding as a result of injections.
Here is research for those science geeks among us. Repurposing Ivermectin for COVID-19: Molecular Aspects and Therapeutic Possibilities
So, to answer the OP, Ivermectin does not kill the spike protein. Something that is not alive to begin with, cannot be killed. The mechanism of action with Ivermectin is targeted to interfere with viral replication within the cell thereby reducing viral load in an actual infection. Ivermectin may interfere with the spike protein binding on ACE2 and CD147 receptors - but these mechanisms still needs further study. Beyond this, Ivermectin could be useful in mitigating cytokine storm resulting from the pathogenic priming caused by the both types of injections.
At the moment, there is nothing that can reverse the production of spike proteins that result from both types of injections for those foolish enough to have received them. All that can be done at this point is to try to lessen the damage and Ivermectin can be used for that purpose. But, Ivermectin does have side effects and should be used with caution as part of a long term strategy to mitigate the effects of injection induced spike protein production.
The specific mechanism of action of the antiparasitic drug Ivermectin as an antiviral is shown to interfere with the viral replication process by inhibiting nuclear importation of viral proteins by its action against the nucleocapsid protein and also by inhibiting RNA dependent RNA polymerase RNA replication. Ivermectin has also been shown to reduce cytokine storm by suppressing production of interleukin 6 and tumor necrosis factor alpha - two major components in the cytokine storm. Some additional research shows that Ivermectin may bind to ACE2 receptors that can interfere with spike protein binding - but this mechanism of action has been seen by computer studies only and has not been shown in actual in vitro or in vivo studies. More computer modeling studies show that Ivermectin can also bind with CD147 receptors on red blood cells and platelets thereby interfering with the binding of spike protein to these blood components that leads to clotting.
Here is research for those science geeks among us. Repurposing Ivermectin for COVID-19: Molecular Aspects and Therapeutic Possibilities
So, to answer the OP, Ivermectin does not kill the spike protein. Something that is not alive to begin with, cannot be killed. The mechanism of action with Ivermectin is targeted to interfere with viral replication within the cell thereby reducing viral load in an actual infection. Ivermectin may interfere with the spike protein binding on ACE2 and CD147 receptors - but these mechanisms still needs further study. Beyond this, Ivermectin could be useful in mitigating cytokine storm resulting from the pathogenic priming caused by the both types of injections.
At the moment, there is nothing that can reverse the production of spike proteins that result from both types of injections for those foolish enough to have received them. All that can be done at this point is to try to lessen the damage and Ivermectin can be used for that purpose. But, Ivermectin does have side effects and should be used with caution as part of a long term strategy to mitigate the effects of injection induced spike protein production.
The specific mechanism of action of the antiparasitic drug Ivermectin as an antiviral is shown to interfere with the viral replication process by inhibiting nuclear importation of viral proteins by its action against the nucleocapsid protein and RNA dependent RNA polymerase inhibition. Ivermectin has also been shown to reduce cytokine storm by suppressing production of interleukin 6 and tumor necrosis factor alpha - two major components in the cytokine storm. Some additional research shows that Ivermectin may bind to ACE2 receptors that can interfere with spike protein binding - but this mechanism of action has been seen by computer studies only and has not been shown in actual in vitro or in vivo studies. More computer modeling studies show that Ivermectin can also bind with CD147 receptors on red blood cells and platelets thereby interfering with the binding of spike protein to these blood components that leads to clotting.
Here is research for those science geeks among us. Repurposing Ivermectin for COVID-19: Molecular Aspects and Therapeutic Possibilities
So, to answer the OP, Ivermectin does not kill the spike protein. Something that is not alive to begin with, cannot be killed. The mechanism of action with Ivermectin is targeted to interfere with viral replication within the cell thereby reducing viral load in an actual infection. Ivermectin may interfere with the spike protein binding on ACE2 and CD147 receptors - but these mechanisms still needs further study. Beyond this, Ivermectin could be useful in mitigating cytokine storm resulting from the pathogenic priming caused by the both types of injections.
At the moment, there is nothing that can reverse the production of spike proteins that result from both types of injections for those foolish enough to have received them. All that can be done at this point is to try to lessen the damage and Ivermectin can be used for that purpose. But, Ivermectin does have side effects and should be used with caution as part of a long term strategy to mitigate the effects of injection induced spike protein production.
The specific mechanism of action of the antiparasitic drug Ivermectin as an antiviral is shown to interfere with the viral replication process by inhibiting nuclear importation of viral proteins by its action against the nucleocapsid protein. Ivermectin has also been shown to reduce cytokine storm by suppressing production of interleukin 6 and tumor necrosis factor alpha - two major components in the cytokine storm. Some additional research shows that Ivermectin may bind to ACE2 receptors that can interfere with spike protein binding - but this mechanism of action has been seen by computer studies only and has not been shown in actual in vitro or in vivo studies. More computer modeling studies show that Ivermectin can also bind with CD147 receptors on red blood cells and platelets thereby interfering with the binding of spike protein to these blood components that leads to clotting.
Here is research for those science geeks among us. Repurposing Ivermectin for COVID-19: Molecular Aspects and Therapeutic Possibilities
So, to answer the OP, Ivermectin does not kill the spike protein. Something that is not alive to begin with, cannot be killed. The mechanism of action with Ivermectin is targeted to interfere with viral replication within the cell thereby reducing viral load in an actual infection. Ivermectin may interfere with the spike protein binding on ACE2 and CD147 receptors - but these mechanisms still needs further study. Beyond this, Ivermectin could be useful in mitigating cytokine storm resulting from the pathogenic priming caused by the both types of injections.
At the moment, there is nothing that can reverse the production of spike proteins that result from both types of injections for those foolish enough to have received them. All that can be done at this point is to try to lessen the damage and Ivermectin can be used for that purpose. But, Ivermectin does have side effects and should be used with caution as part of a long term strategy to mitigate the effects of injection induced spike protein production.
The specific mechanism of action of Ivermectin interferes with the viral replication process by inhibiting nuclear importation of viral proteins by its action against the nucleocapsid protein. Ivermectin has also been shown to reduce cytokine storm by suppressing production of interleukin 6 and tumor necrosis factor alpha - two major components in the cytokine storm. Some additional research shows that Ivermectin may bind to ACE2 receptors that can interfere with spike protein binding - but this mechanism of action has been seen by computer studies only and has not been shown in actual in vitro or in vivo studies. More computer modeling studies show that Ivermectin can also bind with CD147 receptors on red blood cells and platelets thereby interfering with the binding of spike protein to these blood components that leads to clotting.
Here is research for those science geeks among us. Repurposing Ivermectin for COVID-19: Molecular Aspects and Therapeutic Possibilities
So, to answer the OP, Ivermectin does not kill the spike protein. Something that is not alive to begin with, cannot be killed. The mechanism of action with Ivermectin is targeted to interfere with viral replication within the cell thereby reducing viral load in an actual infection. Ivermectin may interfere with the spike protein binding on ACE2 and CD147 receptors - but these mechanisms still needs further study. Beyond this, Ivermectin could be useful in mitigating cytokine storm resulting from the pathogenic priming caused by the both types of injections.
At the moment, there is nothing that can reverse the production of spike proteins that result from both types of injections for those foolish enough to have received them. All that can be done at this point is to try to lessen the damage and Ivermectin can be used for that purpose. But, Ivermectin does have side effects and should be used with caution as part of a long term strategy to mitigate the effects of injection induced spike protein production.
The specific mechanism of action of Ivermectin interferes with the viral replication process by inhibiting nuclear importation of viral proteins by its action against the nucleocapsid protein. Ivermectin has also been shown to reduce cytokine storm by suppressing production of interleukin 6 and tumor necrosis factor alpha - two major components in the cytokine storm. Some additional research shows that Ivermectin may bind to ACE2 receptors that can interfere with spike protein binding - but this mechanism of action has been seen by computer studies only and has not been shown in actual in vitro or in vivo studies.
Here is research for those science geeks among us. Repurposing Ivermectin for COVID-19: Molecular Aspects and Therapeutic Possibilities
So, to answer the OP, Ivermectin does not kill the spike protein. Something that is not alive to begin with, cannot be killed. The mechanism of action with Ivermectin is targeted to interfere with viral replication within the cell thereby reducing viral load in an actual infection. Ivermectin may interfere with the spike protein binding - but this mechanism still needs further study. Beyond this, Ivermectin could be useful in mitigating cytokine storm resulting from the pathogenic priming caused by the both types of injections.
At the moment, there is nothing that can reverse the production of spike proteins that result from both types of injections for those foolish enough to have received them. All that can be done at this point is to try to lessen the damage and Ivermectin can be used for that purpose. But, Ivermectin does have side effects and should be used with caution as part of a long term strategy to mitigate the effects of injection induced spike protein production.
The specific mechanism of action of Ivermectin interferes with the viral replication process by inhibiting nuclear importation of viral proteins by its action against the nucleocapsid protein. Ivermectin has also been shown to reduce cytokine storm by suppressing production of interleukin 6 and tumor necrosis factor alpha - two major components in the cytokine storm. Some additional research shows that Ivermectin may bind to ACE2 receptors that can interfere with spike protein binding - but this mechanism of action has been seen by computer studies only and has not been shown in actual in vitro or in vivo studies.
Here is research for those science geeks among us. Repurposing Ivermectin for COVID-19: Molecular Aspects and Therapeutic Possibilities
So, to answer the OP, Ivermectin does not kill the spike protein. Something that is not alive to begin with, cannot be killed. The mechanism of action with Ivermectin is targeted to interfere with viral replication within the cell thereby reducing viral load in an actual infection. Ivermectin may interfere with the spike protein binding - but this mechanism still needs further study. Beyond this, Ivermectin could be useful in mitigating cytokine storm from pathogenic priming caused by the antibody produced from both types of injections.
At the moment, there is nothing that can reverse the production of spike proteins that result from both types of injections for those foolish enough to have received them. All that can be done at this point is to try to lessen the damage and Ivermectin can be used for that purpose. But, Ivermectin does have side effects and should be used with caution as part of a long term strategy to mitigate the effects of injection induced spike protein production.
The specific mechanism of action of Ivermectin interferes with the viral replication process by inhibiting nuclear importation of viral proteins by its action against the nucleocapsid protein. Ivermectin has also been shown to reduce cytokine storm by suppressing production of interleukin 6 and tumor necrosis factor alpha - two major components in the cytokine storm. Some additional research shows that Ivermectin may bind to ACE2 receptors that can interfere with spike protein binding - but this mechanism of action has been seen by computer studies only and has not been shown in actual in vitro or in vivo studies.
Here is research for those science geeks among us. Repurposing Ivermectin for COVID-19: Molecular Aspects and Therapeutic Possibilities
So, to answer the OP, Ivermectin does not kill the spike protein. Something that is not alive to begin with, cannot be killed. The mechanism of action of Ivermectin is targeted to interfere with viral replication within the cell and has nothing to do with the spike protein. Ivermectin may interfere with the spike protein binding - but this mechanism still needs further study. It can be useful in reducing viral replication in an actual infection with certain viruses. Beyond this, Ivermectin could be useful in mitigating cytokine storm from pathogenic priming caused by the antibody produced from both types of injections.
At the moment, there is nothing that can reverse the production of spike proteins that result from both types of injections for those foolish enough to have received them. All that can be done at this point is to try to lessen the damage and Ivermectin can be used for that purpose. But, Ivermectin does have side effects and should be used with caution as part of a long term strategy to mitigate the effects of injection induced spike protein production.
The specific mechanism of action of Ivermectin interferes with the viral replication process by inhibiting nuclear importation of viral proteins by its action against the nucleocapsid protein. Ivermectin has also been shown to reduce cytokine storm by suppressing production of interleukin 6 and tumor necrosis factor alpha - two major components in the cytokine storm. Some additional research shows that Ivermectin can bind to ACE2 receptors that can interfere with spike protein binding.
Here is research for those science geeks among us. Repurposing Ivermectin for COVID-19: Molecular Aspects and Therapeutic Possibilities
So, to answer the OP, Ivermectin does not kill the spike protein. Something that is not alive to begin with, cannot be killed. The mechanism of action of Ivermectin is targeted to interfere with viral replication within the cell and has nothing to do with the spike protein. But, Ivermectin can interfere with the spike protein binding and can be be useful in reducing viral replication in an actual infection with certain viruses. Beyond this, Ivermectin could be useful in mitigating cytokine storm from pathogenic priming caused by the antibody produced from both types of injections.
At the moment, there is nothing that can reverse the production of spike proteins that result from both types of injections for those foolish enough to have received them. All that can be done at this point is to try to lessen the damage and Ivermectin can be used for that purpose. But, Ivermectin does have side effects and should be used with caution as part of a long term strategy to mitigate the effects of injection induced spike protein production.
The specific mechanism of action of Ivermectin interferes with the viral replication process by inhibiting nuclear importation of viral proteins by its action against the nucleocapsid protein. Ivermectin has also been shown to reduce cytokine storm by suppressing production of interleukin 6 and tumor necrosis factor alpha - two major components in the cytokine storm. Some additional research shows that Ivermectin can bind to ACE2 receptors that can interfere with spike protein binding.
Here is research for those science geeks among us. Repurposing Ivermectin for COVID-19: Molecular Aspects and Therapeutic Possibilities
So, to answer the OP, Ivermectin does not kill the spike protein. Something that is not alive to begin with, cannot be killed. The mechanism of action of Ivermectin is targeted to interfere with viral replication within the cell and has nothing to do with the spike protein. But, Ivermectin can interfere with the spike protein binding and can be be useful in reducing viral replication in an actual infection with certain viruses. Beyond this, Ivermectin could be useful in mitigating cytokine storm from pathogenic priming caused by the antibody produced from both types of injections.
The specific mechanism of action of Ivermectin interferes with the viral replication process by inhibiting nuclear importation of viral proteins by its action against the nucleocapsid protein. Also, Ivermectin has been shown to reduce cytokine storm by suppressing production of interleukin 6 and tumor necrosis factor alpha - two major components in the cytokine storm. Here is research for those science geeks among us. Repurposing Ivermectin for COVID-19: Molecular Aspects and Therapeutic Possibilities
So, to answer the OP, Ivermectin does not kill the spike protein. Something that is not alive to begin with, cannot be killed. The mechanism of action of Ivermectin is targeted to interfere with viral replication within the cell and has nothing to do with the spike protein. Ivermectin would only be useful in reducing viral replication in an actual infection with certain viruses. Beyond this, Ivermectin could be useful in mitigating cytokine storm from pathogenic priming caused by the antibody produced from both types of injections.