"Pauling said arterial plaque was the result of the body trying to repair damage caused by a long-term vitamin C deficiency. He believed heart disease was a form of low-level scurvy (yes, the disease that used to strike sailors of old on long ocean voyages) and that the plaque was due to the body reinforcing and patching up weakened areas of the blood vessels that would otherwise rupture.

Pauling also showed that heart disease could be prevented or treated by taking vitamin C and other supplements. Just as carrying limes and other citrus fruits on ships became the norm when we found out it stopped the dreaded scurvy.

Pauling’s Revolutionary Theory Was Developed On Several Important Scientific Findings: FIRSTLY: Plaque deposits found in human aortas are made from a special form of cholesterol called Lipoprotein a or Lp(a), not from ordinary LDL cholesterol. Lp(a) is a special form of cholesterol that forms the thick sheets of plaque that obstruct arteries.

SECONDLY: Plaque deposits are not deposited uniformly around the circulatory system, instead, they only occur at sites near the heart, on bends, and where mechanical pressure is great. These are points where the artery gets stretched and bent with every heartbeat.

AND LASTLY: That plaque only forms in parts of the artery that are damaged from this constant bending and stretching. As tiny cracks appear in the artery wall, Lysine a component of collagen is slowly released. This lysine attracts Lp(a) to the site of the crack and as time progresses the sheets of Lp(a) deposit on top of each other and start to develop into a dangerous blockage.

As Pauling observed the process of plaque formation, which was similar to that of scurvy, he also noticed other clues that support his theory.

#1. ATHEROSCLEROSIS DOES NOT OCCUR IN ANY ANIMALS THAT PRODUCE THEIR OWN SUPPLY OF VITAMIN C.

#2. ONLY ANIMALS THAT DON’T PRODUCE VITAMIN C ARE ABLE TO PRODUCE LPa. THAT’S HUMANS, APES, AND GUINEA PIGS

PLEASE STOP RIGHT NOW TO CONSIDER THOSE TWO POINTS! They show the direct link between Vitamin C deficiency and Heart Disease! There have also been animal studies that have been found to support his theory. Studies on animals such as guinea pigs which cannot produce their own Vitamin C show that when dietary vitamin C levels are restricted, collagen production drops, and the blood vessels become weaker and thinner. At the same time, the animal begins to increase the blood levels of Lp(a) and plaque is seen to form at points of potential rupture and damage."

Copied from: https://t.co/7K5w1otPa3

Disclaimer: I am also a researcher, not a doctor, so we have something in common.

BTW my husband and I both take 10,000 IU's of Vitamin D daily with a Super K from Life Extension. We recently had heart scans and his calcium score was very high. Mine was higher than it should be. We maintain about 65 ng/mL of D so I was surprised that we would have this problem. Because of the info above, our arteries are constantly patching up the lesions so that the K is not able to keep up at those sites. You need to heal the underlying sub-clinical scurvy. The other post is a gold mine of info about all this and it would be worth looking at.

Would love your feedback on all the Vitamin info links...

"Pauling said arterial plaque was the result of the body trying to repair damage caused by a long-term vitamin C deficiency. He believed heart disease was a form of low-level scurvy (yes, the disease that used to strike sailors of old on long ocean voyages) and that the plaque was due to the body reinforcing and patching up weakened areas of the blood vessels that would otherwise rupture.

Pauling also showed that heart disease could be prevented or treated by taking vitamin C and other supplements. Just as carrying limes and other citrus fruits on ships became the norm when we found out it stopped the dreaded scurvy.

Pauling’s Revolutionary Theory Was Developed On Several Important Scientific Findings: FIRSTLY: Plaque deposits found in human aortas are made from a special form of cholesterol called Lipoprotein a or Lp(a), not from ordinary LDL cholesterol. Lp(a) is a special form of cholesterol that forms the thick sheets of plaque that obstruct arteries.

SECONDLY: Plaque deposits are not deposited uniformly around the circulatory system, instead, they only occur at sites near the heart, on bends, and where mechanical pressure is great. These are points where the artery gets stretched and bent with every heartbeat.

AND LASTLY: That plaque only forms in parts of the artery that are damaged from this constant bending and stretching. As tiny cracks appear in the artery wall, Lysine a component of collagen is slowly released. This lysine attracts Lp(a) to the site of the crack and as time progresses the sheets of Lp(a) deposit on top of each other and start to develop into a dangerous blockage.

As Pauling observed the process of plaque formation, which was similar to that of scurvy, he also noticed other clues that support his theory.

#1. ATHEROSCLEROSIS DOES NOT OCCUR IN ANY ANIMALS THAT PRODUCE THEIR OWN SUPPLY OF VITAMIN C.

#2. ONLY ANIMALS THAT DON’T PRODUCE VITAMIN C ARE ABLE TO PRODUCE LPa. THAT’S HUMANS, APES, AND GUINEA PIGS

PLEASE STOP RIGHT NOW TO CONSIDER THOSE TWO POINTS! They show the direct link between Vitamin C deficiency and Heart Disease! There have also been animal studies that have been found to support his theory. Studies on animals such as guinea pigs which cannot produce their own Vitamin C show that when dietary vitamin C levels are restricted, collagen production drops, and the blood vessels become weaker and thinner. At the same time, the animal begins to increase the blood levels of Lp(a) and plaque is seen to form at points of potential rupture and damage."

Copied from: https://t.co/7K5w1otPa3

Disclaimer: I am also a researcher, not a doctor, so we have something in common.

BTW my husband and I both take 10,000 IU's of Vitamin D daily with a Super K from Life Extension. We recently had heart scans and his calcium score was very high. Mine was higher than it should be. We maintain about 65 ng/mL of D so I was surprised that we would have this problem. Because of the info above, our arteries are constantly patching up the lesions so that the K is not able to keep up at those sites. You need to heal the underlying sub-clinical scurvy. The other post is a gold mine of info about all this and it would be worth looking at.

Pauling said arterial plaque was the result of the body trying to repair damage caused by a long-term vitamin C deficiency. He believed heart disease was a form of low-level scurvy (yes, the disease that used to strike sailors of old on long ocean voyages) and that the plaque was due to the body reinforcing and patching up weakened areas of the blood vessels that would otherwise rupture.

Pauling also showed that heart disease could be prevented or treated by taking vitamin C and other supplements. Just as carrying limes and other citrus fruits on ships became the norm when we found out it stopped the dreaded scurvy.

Pauling’s Revolutionary Theory Was Developed On Several Important Scientific Findings: FIRSTLY: Plaque deposits found in human aortas are made from a special form of cholesterol called Lipoprotein a or Lp(a), not from ordinary LDL cholesterol. Lp(a) is a special form of cholesterol that forms the thick sheets of plaque that obstruct arteries.

SECONDLY: Plaque deposits are not deposited uniformly around the circulatory system, instead, they only occur at sites near the heart, on bends, and where mechanical pressure is great. These are points where the artery gets stretched and bent with every heartbeat.

AND LASTLY: That plaque only forms in parts of the artery that are damaged from this constant bending and stretching. As tiny cracks appear in the artery wall, Lysine a component of collagen is slowly released. This lysine attracts Lp(a) to the site of the crack and as time progresses the sheets of Lp(a) deposit on top of each other and start to develop into a dangerous blockage.

As Pauling observed the process of plaque formation, which was similar to that of scurvy, he also noticed other clues that support his theory.

#1. ATHEROSCLEROSIS DOES NOT OCCUR IN ANY ANIMALS THAT PRODUCE THEIR OWN SUPPLY OF VITAMIN C.

#2. ONLY ANIMALS THAT DON’T PRODUCE VITAMIN C ARE ABLE TO PRODUCE LPa. THAT’S HUMANS, APES, AND GUINEA PIGS

PLEASE STOP RIGHT NOW TO CONSIDER THOSE TWO POINTS! They show the direct link between Vitamin C deficiency and Heart Disease! There have also been animal studies that have been found to support his theory. Studies on animals such as guinea pigs which cannot produce their own Vitamin C show that when dietary vitamin C levels are restricted, collagen production drops, and the blood vessels become weaker and thinner. At the same time, the animal begins to increase the blood levels of Lp(a) and plaque is seen to form at points of potential rupture and damage.

Disclaimer: I am also a researcher, not a doctor, so we have something in common.

BTW my husband and I both take 10,000 IU's of Vitamin D daily with a Super K from Life Extension. We recently had heart scans and his calcium score was very high. Mine was higher than it should be. We maintain about 65 ng/mL of D so I was surprised that we would have this problem. Because of the info above, our arteries are constantly patching up the lesions so that the K is not able to keep up at those sites. You need to heal the underlying sub-clinical scurvy. The other post is a gold mine of info about all this and it would be worth looking at.

Pauling said arterial plaque was the result of the body trying to repair damage caused by a long-term vitamin C deficiency. He believed heart disease was a form of low-level scurvy (yes, the disease that used to strike sailors of old on long ocean voyages) and that the plaque was due to the body reinforcing and patching up weakened areas of the blood vessels that would otherwise rupture.

Pauling also showed that heart disease could be prevented or treated by taking vitamin C and other supplements. Just as carrying limes and other citrus fruits on ships became the norm when we found out it stopped the dreaded scurvy.

Pauling’s Revolutionary Theory Was Developed On Several Important Scientific Findings: FIRSTLY: Plaque deposits found in human aortas are made from a special form of cholesterol called Lipoprotein a or Lp(a), not from ordinary LDL cholesterol. Lp(a) is a special form of cholesterol that forms the thick sheets of plaque that obstruct arteries.

SECONDLY: Plaque deposits are not deposited uniformly around the circulatory system, instead, they only occur at sites near the heart, on bends, and where mechanical pressure is great. These are points where the artery gets stretched and bent with every heartbeat.

AND LASTLY: That plaque only forms in parts of the artery that are damaged from this constant bending and stretching. As tiny cracks appear in the artery wall, Lysine a component of collagen is slowly released. This lysine attracts Lp(a) to the site of the crack and as time progresses the sheets of Lp(a) deposit on top of each other and start to develop into a dangerous blockage.

As Pauling observed the process of plaque formation, which was similar to that of scurvy, he also noticed other clues that support his theory.

#1. ATHEROSCLEROSIS DOES NOT OCCUR IN ANY ANIMALS THAT PRODUCE THEIR OWN SUPPLY OF VITAMIN C.

#2. ONLY ANIMALS THAT DON’T PRODUCE VITAMIN C ARE ABLE TO PRODUCE LPa. THAT’S HUMANS, APES, AND GUINEA PIGS

PLEASE STOP RIGHT NOW TO CONSIDER THOSE TWO POINTS! They show the direct link between Vitamin C deficiency and Heart Disease! There have also been animal studies that have been found to support his theory. Studies on animals such as guinea pigs which cannot produce their own Vitamin C show that when dietary vitamin C levels are restricted, collagen production drops, and the blood vessels become weaker and thinner. At the same time, the animal begins to increase the blood levels of Lp(a) and plaque is seen to form at points of potential rupture and damage.

Disclaimer: I am also a researcher, not a doctor, so we have something in common.

BTW my husband and I both take 10,000 IU's of Vitamin D daily with a Super K from Life Extension. We recently had heart scans and his calcium score was very high. Mine was higher than it should be. We maintain about 65 ng/mL of D so I was surprised that we would have this problem. Because of the info above, our arteries are constantly patching up the lesions so that the K is not able to work properly. You need to heal the underlying sub-clinical scurvy. The other post is a gold mine of info about all this and it would be worth looking at.

Pauling said arterial plaque was the result of the body trying to repair damage caused by a long-term vitamin C deficiency. He believed heart disease was a form of low-level scurvy (yes, the disease that used to strike sailors of old on long ocean voyages) and that the plaque was due to the body reinforcing and patching up weakened areas of the blood vessels that would otherwise rupture.

Pauling also showed that heart disease could be prevented or treated by taking vitamin C and other supplements. Just as carrying limes and other citrus fruits on ships became the norm when we found out it stopped the dreaded scurvy.

Pauling’s Revolutionary Theory Was Developed On Several Important Scientific Findings: FIRSTLY: Plaque deposits found in human aortas are made from a special form of cholesterol called Lipoprotein a or Lp(a), not from ordinary LDL cholesterol. Lp(a) is a special form of cholesterol that forms the thick sheets of plaque that obstruct arteries.

SECONDLY: Plaque deposits are not deposited uniformly around the circulatory system, instead, they only occur at sites near the heart, on bends, and where mechanical pressure is great. These are points where the artery gets stretched and bent with every heartbeat.

AND LASTLY: That plaque only forms in parts of the artery that are damaged from this constant bending and stretching. As tiny cracks appear in the artery wall, Lysine a component of collagen is slowly released. This lysine attracts Lp(a) to the site of the crack and as time progresses the sheets of Lp(a) deposit on top of each other and start to develop into a dangerous blockage.

As Pauling observed the process of plaque formation, which was similar to that of scurvy, he also noticed other clues that support his theory.

#1. ATHEROSCLEROSIS DOES NOT OCCUR IN ANY ANIMALS THAT PRODUCE THEIR OWN SUPPLY OF VITAMIN C.

#2. ONLY ANIMALS THAT DON’T PRODUCE VITAMIN C ARE ABLE TO PRODUCE LPa. THAT’S HUMANS, APES, AND GUINEA PIGS

PLEASE STOP RIGHT NOW TO CONSIDER THOSE TWO POINTS! They show the direct link between Vitamin C deficiency and Heart Disease! There have also been animal studies that have been found to support his theory. Studies on animals such as guinea pigs which cannot produce their own Vitamin C show that when dietary vitamin C levels are restricted, collagen production drops, and the blood vessels become weaker and thinner. At the same time, the animal begins to increase the blood levels of Lp(a) and plaque is seen to form at points of potential rupture and damage.

Disclaimer: I am also a researcher, not a doctor, so we have something in common.

BTW my husband and I both take 10,000 IU's of Vitamin D daily with a Super K from Life Extension. We recently had heart scans and his calcium score was very high. Mine was higher than it should be. We maintain about 65 ng/mL of D so I was surprised that we would have this problem. Because of the info above, our afteries are constantly patching up the lesions so that the K is not able to work properly. You need to heal the underlying sub-clinical scurvy. The other post is a gold mine of info about all this and it would be worth looking at.

Pauling said arterial plaque was the result of the body trying to repair damage caused by a long-term vitamin C deficiency. He believed heart disease was a form of low-level scurvy (yes, the disease that used to strike sailors of old on long ocean voyages) and that the plaque was due to the body reinforcing and patching up weakened areas of the blood vessels that would otherwise rupture.

Pauling also showed that heart disease could be prevented or treated by taking vitamin C and other supplements. Just as carrying limes and other citrus fruits on ships became the norm when we found out it stopped the dreaded scurvy.

Pauling’s Revolutionary Theory Was Developed On Several Important Scientific Findings: FIRSTLY: Plaque deposits found in human aortas are made from a special form of cholesterol called Lipoprotein a or Lp(a), not from ordinary LDL cholesterol. Lp(a) is a special form of cholesterol that forms the thick sheets of plaque that obstruct arteries.

SECONDLY: Plaque deposits are not deposited uniformly around the circulatory system, instead, they only occur at sites near the heart, on bends, and where mechanical pressure is great. These are points where the artery gets stretched and bent with every heartbeat.

AND LASTLY: That plaque only forms in parts of the artery that are damaged from this constant bending and stretching. As tiny cracks appear in the artery wall, Lysine a component of collagen is slowly released. This lysine attracts Lp(a) to the site of the crack and as time progresses the sheets of Lp(a) deposit on top of each other and start to develop into a dangerous blockage.

As Pauling observed the process of plaque formation, which was similar to that of scurvy, he also noticed other clues that support his theory.

#1. ATHEROSCLEROSIS DOES NOT OCCUR IN ANY ANIMALS THAT PRODUCE THEIR OWN SUPPLY OF VITAMIN C.

#2. ONLY ANIMALS THAT DON’T PRODUCE VITAMIN C ARE ABLE TO PRODUCE LPa. THAT’S HUMANS, APES, AND GUINEA PIGS

PLEASE STOP RIGHT NOW TO CONSIDER THOSE TWO POINTS! They show the direct link between Vitamin C deficiency and Heart Disease! There have also been animal studies that have been found to support his theory. Studies on animals such as guinea pigs which cannot produce their own Vitamin C show that when dietary vitamin C levels are restricted, collagen production drops, and the blood vessels become weaker and thinner. At the same time, the animal begins to increase the blood levels of Lp(a) and plaque is seen to form at points of potential rupture and damage.

Disclaimer: I am also a researcher, not a doctor, so we have something in common.

BTW my husband and I both take 10,000 IU's of Vitamin D daily with a Super K from Life Extension. We recently had heart scans and his calcium score was very high. Mine was higher than it should be. We maintain about 65 ng/mL of D so I was surprised that we would have this problem. Because of the info above, our afteries are constantly patching up the lesions so that the K is not able to work properly. You need to heal the underlying sub-clinical scurvy. The other post is a gold mine of info about all this and it would be worth looking at.

Pauling said arterial plaque was the result of the body trying to repair damage caused by a long-term vitamin C deficiency. He believed heart disease was a form of low-level scurvy (yes, the disease that used to strike sailors of old on long ocean voyages) and that the plaque was due to the body reinforcing and patching up weakened areas of the blood vessels that would otherwise rupture.

Pauling also showed that heart disease could be prevented or treated by taking vitamin C and other supplements. Just as carrying limes and other citrus fruits on ships became the norm when we found out it stopped the dreaded scurvy.

Pauling’s Revolutionary Theory Was Developed On Several Important Scientific Findings: FIRSTLY: Plaque deposits found in human aortas are made from a special form of cholesterol called Lipoprotein a or Lp(a), not from ordinary LDL cholesterol. Lp(a) is a special form of cholesterol that forms the thick sheets of plaque that obstruct arteries.

SECONDLY: Plaque deposits are not deposited uniformly around the circulatory system, instead, they only occur at sites near the heart, on bends, and where mechanical pressure is great. These are points where the artery gets stretched and bent with every heartbeat.

AND LASTLY: That plaque only forms in parts of the artery that are damaged from this constant bending and stretching. As tiny cracks appear in the artery wall, Lysine a component of collagen is slowly released. This lysine attracts Lp(a) to the site of the crack and as time progresses the sheets of Lp(a) deposit on top of each other and start to develop into a dangerous blockage.

As Pauling observed the process of plaque formation, which was similar to that of scurvy, he also noticed other clues that support his theory.

#1. ATHEROSCLEROSIS DOES NOT OCCUR IN ANY ANIMALS THAT PRODUCE THEIR OWN SUPPLY OF VITAMIN C.

#2. ONLY ANIMALS THAT DON’T PRODUCE VITAMIN C ARE ABLE TO PRODUCE LPa. THAT’S HUMANS, APES, AND GUINEA PIGS

PLEASE STOP RIGHT NOW TO CONSIDER THOSE TWO POINTS! They show the direct link between Vitamin C deficiency and Heart Disease! There have also been animal studies that have been found to support his theory. Studies on animals such as guinea pigs which cannot produce their own Vitamin C show that when dietary vitamin C levels are restricted, collagen production drops, and the blood vessels become weaker and thinner. At the same time, the animal begins to increase the blood levels of Lp(a) and plaque is seen to form at points of potential rupture and damage.

Disclaimer: I am also a researcher, not a doctor, so we have something in common.

BTW my husband and I both take 10,000 IU's of Vitamin D daily with a Super K from Life Extension. We recently had heart scans and his calcium score was very high. Mine was higher than it should be. We maintain about 65 ng/mL so I was surprised that we would have this problem. Because of the info above, our afteries are constantly patching up the lesions so that the K is not able to work properly. You need to heal the underlying sub-clinical scurvy. The other post is a gold mine of info about all this and it would be worth looking at.

Pauling said arterial plaque was the result of the body trying to repair damage caused by a long-term vitamin C deficiency. He believed heart disease was a form of low-level scurvy (yes, the disease that used to strike sailors of old on long ocean voyages) and that the plaque was due to the body reinforcing and patching up weakened areas of the blood vessels that would otherwise rupture.

Pauling also showed that heart disease could be prevented or treated by taking vitamin C and other supplements. Just as carrying limes and other citrus fruits on ships became the norm when we found out it stopped the dreaded scurvy.

Pauling’s Revolutionary Theory Was Developed On Several Important Scientific Findings: FIRSTLY: Plaque deposits found in human aortas are made from a special form of cholesterol called Lipoprotein a or Lp(a), not from ordinary LDL cholesterol. Lp(a) is a special form of cholesterol that forms the thick sheets of plaque that obstruct arteries.

SECONDLY: Plaque deposits are not deposited uniformly around the circulatory system, instead, they only occur at sites near the heart, on bends, and where mechanical pressure is great. These are points where the artery gets stretched and bent with every heartbeat.

AND LASTLY: That plaque only forms in parts of the artery that are damaged from this constant bending and stretching. As tiny cracks appear in the artery wall, Lysine a component of collagen is slowly released. This lysine attracts Lp(a) to the site of the crack and as time progresses the sheets of Lp(a) deposit on top of each other and start to develop into a dangerous blockage.

As Pauling observed the process of plaque formation, which was similar to that of scurvy, he also noticed other clues that support his theory.

#1. ATHEROSCLEROSIS DOES NOT OCCUR IN ANY ANIMALS THAT PRODUCE THEIR OWN SUPPLY OF VITAMIN C.

#2. ONLY ANIMALS THAT DON’T PRODUCE VITAMIN C ARE ABLE TO PRODUCE LPa. THAT’S HUMANS, APES, AND GUINEA PIGS

PLEASE STOP RIGHT NOW TO CONSIDER THOSE TWO POINTS! They show the direct link between Vitamin C deficiency and Heart Disease! There have also been animal studies that have been found to support his theory. Studies on animals such as guinea pigs which cannot produce their own Vitamin C show that when dietary vitamin C levels are restricted, collagen production drops, and the blood vessels become weaker and thinner. At the same time, the animal begins to increase the blood levels of Lp(a) and plaque is seen to form at points of potential rupture and damage.

Disclaimer: I am also a researcher, not a doctor, so we have something in common.

Pauling said arterial plaque was the result of the body trying to repair damage caused by a long-term vitamin C deficiency. He believed heart disease was a form of low-level scurvy (yes, the disease that used to strike sailors of old on long ocean voyages) and that the plaque was due to the body reinforcing and patching up weakened areas of the blood vessels that would otherwise rupture.

Pauling also showed that heart disease could be prevented or treated by taking vitamin C and other supplements. Just as carrying limes and other citrus fruits on ships became the norm when we found out it stopped the dreaded scurvy.

Pauling’s Revolutionary Theory Was Developed On Several Important Scientific Findings: FIRSTLY: Plaque deposits found in human aortas are made from a special form of cholesterol called Lipoprotein a or Lp(a), not from ordinary LDL cholesterol. Lp(a) is a special form of cholesterol that forms the thick sheets of plaque that obstruct arteries.

SECONDLY: Plaque deposits are not deposited uniformly around the circulatory system, instead, they only occur at sites near the heart, on bends, and where mechanical pressure is great. These are points where the artery gets stretched and bent with every heartbeat.

AND LASTLY: That plaque only forms in parts of the artery that are damaged from this constant bending and stretching. As tiny cracks appear in the artery wall, Lysine a component of collagen is slowly released. This lysine attracts Lp(a) to the site of the crack and as time progresses the sheets of Lp(a) deposit on top of each other and start to develop into a dangerous blockage.

As Pauling observed the process of plaque formation, which was similar to that of scurvy, he also noticed other clues that support his theory.

#1. ATHEROSCLEROSIS DOES NOT OCCUR IN ANY ANIMALS THAT PRODUCE THEIR OWN SUPPLY OF VITAMIN C.

#2. ONLY ANIMALS THAT DON’T PRODUCE VITAMIN C ARE ABLE TO PRODUCE LPa. THAT’S HUMANS, APES, AND GUINEA PIGS

PLEASE STOP RIGHT NOW TO CONSIDER THOSE TWO POINTS! They show the direct link between Vitamin C deficiency and Heart Disease! There have also been animal studies that have been found to support his theory. Studies on animals such as guinea pigs which cannot produce their own Vitamin C show that when dietary vitamin C levels are restricted, collagen production drops, and the blood vessels become weaker and thinner. At the same time, the animal begins to increase the blood levels of Lp(a) and plaque is seen to form at points of potential rupture and damage.

Disclaimer: I am also a researcher, not a doctor, so we have something in common.