Thank you for your detailed response and insights. I was just throwing around ideas to see if any would stick. Indeed, it would seem logical that a cell presenting the spike proteins would trigger the same kind of immune response as would the virus itself, though I don’t know if this is exactly the case. In which case, any problems regarding the immune system attacking parts of the human body due to the spike protein-learning would likely also occur from natural encounters and learning of the virus itself. Which is to say, it would not happen (if it did, it would be a fatal flaw in the design of the immune system, which I highly doubt).

I don’t have the same reading that you do, so hearing that the similarity between the spike protein and reproductive protein is limited to a sequence of 4 amino acids is very reassuring. I assume even this small matchup is very uncommon, otherwise it would not have been brought up to attention in the first place, but it sounds like this is still of low concern.

The topic of cytokine storm is interesting, though I need to look into how that situation even occurs to begin with. My understanding of it is that the immune system configures itself in a certain way in response to the presented protein, which somehow disrupts the whole start & stop mechanism. If that primitive understanding is correct, then it begs the question of how come this same situation does not also happen as a result of natural encounters with the virus? Or maybe it does?