If you recall, the trouble with these so-called "vaccines" was that the test subjects were fine for a while, but when exposed to another virus they all went into unrecoverable cytokine storms and died. I know most of us know a lot of people who have been injected with these experimental gene therapies. Most of them don't know the lab history of them.
So while "MesenCure" can help patients with Covid and pneumonia (that's all they are talking about with it right now), I'm really more interested in the possibility that all of these people who were vaxxed can be helped down the road when the inevitable happens.
That would be the effects of ADE... but how long does that last as a result of these injections? There are papers circulating that suggest that reverse transcriptase could/would make the production of the spike proteins permanent... (no proof of that though).
There are numerous biological barriers that prevent mRNA from writing to DNA. It isn't impossible, but its an extraordinarily rare event. In the case of some viruses, like HIV the virus itself causes the cell to express a viral reverse transcriptase (RT). Even then it rarely writes its RNA to the cells DNA. Once the mRNA leaves the nucleus its out. Its a one way trip. Or in the case of a RNA virus it's never even gets into the nucleus.
When a cell undergoes mitosis and the nuclear envelope breaks down it can potentially get written then, but only if a RT is also expressed. And again, even then its rare since it would have to do it in a way that gets past the various protections and checkpoints that occur during mitosis to prevent that event. The cell really doesn't want that to happen, so its very rare.
In a case like the mRNA immunotherapies there is no RT expression. Cells don't just express RT's willy nilly. The ones that are expressed in cells only occur at specific times and only write specific sequences of RNA (such as telomerase).
The "paper" (which was actually a non reviewed pre-print) caused the viral RNA (not the mRNA from the vaccines, but the actual virus, with all its associated proteins) to be written when coexpressing exogenous RT's (from additional transfection into the cell).
They did get a signal without the exogenous RTs, but it was during induced mitosis (which is something the cell would not normally do under viral load) and the signal was so small it was within the error bars. So it was actually a rather poor paper and I doubt it will pass peer review (even if there was no fraudulent incentive to keep such things out of the literature).
I'm not sure how long the potential for an ADE effect from pathogenic priming might be a risk. I don't know enough about the pathways involved. I need to do more research on it. If I had to guess it would be a year or less. I think its an effect of the short term immune system and not long term memory T-cells. But I really haven't done my due diligence on that yet. Too many things to worry about.
I have seen reason to think that the effect can be avoided by the same systems that fight the SARS virus itself (Vitamin D, C, Zn, HCQ, Ivermectin), so its not high on my list of concerns. In addition, I think if it was a really bad problem we would already know.