I appreciate that post. Thank you for that.

The issue with the study is that we are diurnal animals and mice are nocturnal animals. What does that mean? It means a weak vs a strong covalent bonding with melanopsin/Vitamin A and that relates to how our vitamin a cycle - and tadahhhh how our melatonin cycle works.

Studies done on mice in this regard are worthless. Because of the covalent bonding between melanopsin and retinol.

But, the endocabbinoid system links to the immune system which is why it works - IN SOME CASES - not ALL cases.

See, when electron chain transport is fast - apoptosis can't happen (= when cancer grows). What slows down ECT? Endocannabinoids. What also slows down ECT? Melatonin. See how that works?

In nature we wouldn't need the cannabis, but cannabis can help you regain apoptosis, because it slows down electron chain transport.

Now, how do you best build your cannabinoid system?

What programs your immune system? (T-cells etc.) Light. So it's back to where we started having this conversation.

Thanks for sharing that.