There was a paper about this in February this year.

they are talking about long covid (as opposed to covid, which is not the same disease).

Anyway, the clots are fibrin clots, due to platelet hyperactivation. One can pick it up with unusually high D-dimer tests apparently - so if one is feeling breathlessness, fatigue, chest pain, myalgia, cognitive dysfunction, innate immune responses, long after the bug has gone away, then it may be time to request a d-dimer test.and although TEG assays are pretty useless, because the fibrin has already coagulated, they can still be useful.

While we do not yet know the details of the mechanical properties of the fibrin amyloid microclots, it is known that amyloid fibrils can typically exhibit unusually high mechanical strength and resistance to deformation (e.g. [174–177]. The stiffness also increases with the thickness of the fibres [178]. This implies strongly that the fibrinaloids are likely to be more prone to ‘getting stuck’ in capillaries.

Here are comorbidities that will increase the likelihood:

Alzheimer's [37,50,60–62], Parkinson's disease [37,48], Type 2 diabetes [37,38,62,63,213,214] (where the amyloid protein amylin [215–217] is of course a well-known player) and rheumatoid arthritis [65,67].

Others are: Dengue [218–220], Ebola [221–223], Lyme [224], Zika

they think that high iron levels may help, because covid apperntly rips iron out of cells. Iron chelates the fibrin structures.

Anyway, this is the protocl they suggest:

Long COVID patients might be treated by one month of dual antiplatelet therapy (DAPT) (Clopidogrel 75 mg/Aspirin 75 mg) once a day, as well as a direct oral anticoagulant (DOAC) (Apixiban) 5 mg twice a day, together with a proton pump inhibitor (PPI) (e.g. pantoprazole 40 mg/day for gastric protection).

I am just an autist: maybe a doctor-pede can check it out here: https://portlandpress.com/biochemj/article/479/4/537/230829/A-central-role-for-amyloid-fibrin-microclots-in