The Fenbendazole and perhaps other anthelmintics basically ‘starve’ the cancer cells by inhibiting their uptake of sugar which is required for their metabolic function. How do I know this?
My 32 yr old friend was diagnosed with colon cancer with metastasis into lymph nodes and other areas. He had a chemo port installed and at last minute changed his mind and started a FB protocol 222mg active daily for 3 months. When he was to go back in for followup PETscan they told him he had to stop the FB 3 days before the test (otherwise it wouldn’t work.!). Why? Because the FB inhibits the uptake of the irradiated sugar used in the test they told him. So, if they know this is the case and that the FB basically is ‘starving’ the cancer - why isn’t this the recommended treatment/cure.? #fuckingcriminals
His PETscan came back cancer-free. They removed the chemo port. While I was ecstatic that it worked I am also saddened that they obviously know what’s going on….and still millions are dying and suffering daily.
Not only that, it also inhibits p-gp.
P-gp is overexpressed in tumor cells, contributing to multidrug resistance (MDR). By pumping drugs out of cancer cells, P-gp can reduce the drugs' effectiveness, making the cancer harder to treat. Check out this diagram I created. Fenbendazole is near the bottom. https://i.ibb.co/5M43BP0/Lung-Cancer.png
I'm sorry, I'm having trouble following your posts. Are you saying taking Fenbendozole makes it harder for chemo to do it's job?? If my chemo treatments are every two weeks, I'm thinking of supplementing the "off" weeks with ivermectin and/or fenbendazole. (stage 4 colon cancer in the liver)
TLDR: No, it will help fight the cancer while allowing chemo to do its job.
Each of these treatment approaches targets cancer cells through different mechanisms:
Chemotherapy primarily disrupts DNA processes
Fenbendazole may interfere with cancer cell metabolism and support immune function by reducing the cancer cells ability to remove unwanted substances.
Targeted therapies and immunotherapies focus on specific cancer cell characteristics or enhancing the immune response, respectively.
Phytochemicals can have various effects, including influencing drug resistance mechanisms and signaling pathways. Combining these strategies can potentially improve treatment outcomes.
If anyone wanna know exactly why this works I can give you details
So can I.
The Fenbendazole and perhaps other anthelmintics basically ‘starve’ the cancer cells by inhibiting their uptake of sugar which is required for their metabolic function. How do I know this?
My 32 yr old friend was diagnosed with colon cancer with metastasis into lymph nodes and other areas. He had a chemo port installed and at last minute changed his mind and started a FB protocol 222mg active daily for 3 months. When he was to go back in for followup PETscan they told him he had to stop the FB 3 days before the test (otherwise it wouldn’t work.!). Why? Because the FB inhibits the uptake of the irradiated sugar used in the test they told him. So, if they know this is the case and that the FB basically is ‘starving’ the cancer - why isn’t this the recommended treatment/cure.? #fuckingcriminals
His PETscan came back cancer-free. They removed the chemo port. While I was ecstatic that it worked I am also saddened that they obviously know what’s going on….and still millions are dying and suffering daily.
Not only that, it also inhibits p-gp. P-gp is overexpressed in tumor cells, contributing to multidrug resistance (MDR). By pumping drugs out of cancer cells, P-gp can reduce the drugs' effectiveness, making the cancer harder to treat. Check out this diagram I created. Fenbendazole is near the bottom. https://i.ibb.co/5M43BP0/Lung-Cancer.png
I'm sorry, I'm having trouble following your posts. Are you saying taking Fenbendozole makes it harder for chemo to do it's job?? If my chemo treatments are every two weeks, I'm thinking of supplementing the "off" weeks with ivermectin and/or fenbendazole. (stage 4 colon cancer in the liver)
Each of these treatment approaches targets cancer cells through different mechanisms:
Chemotherapy primarily disrupts DNA processes
Fenbendazole may interfere with cancer cell metabolism and support immune function by reducing the cancer cells ability to remove unwanted substances.
Targeted therapies and immunotherapies focus on specific cancer cell characteristics or enhancing the immune response, respectively.
Phytochemicals can have various effects, including influencing drug resistance mechanisms and signaling pathways. Combining these strategies can potentially improve treatment outcomes.