Interestingly, it has been shown that HSV-1 spike protein binds to heparin and increases the aggregation of amyloid β (Aβ42) peptides on its surface spikes [11]. This study suggests that the heparin-binding site of the spike protein might act as a binding site for Aβ42 peptides and thus could dock to the viral surface and catalyze aggregation of Aβ42. As the receptor binding domain (RBD) of SARS-CoV-2, which is located within the S1 subunit of spike glycoprotein has several heparin binding sites [[12], [13], [14]], the same mechanism of aggregation of neurodegeneration causing proteins such as Aβ, α-synuclein, tau, prions, and TDP-43 can be observed in COVID-19 infection in the brain.
Spez: Alternatively, Ctrl-F Prion without the s and you get 21 hits.
Whoa thanks for posting this. Bookmarked. This lends me to believe JohnHereToHelp on this subject. I was beginning to grow suspicious of him but maybe he is really telling the truth.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7988450/ Holy shit.
Ctrl-F Prions. The first result reads:
Spez: Alternatively, Ctrl-F Prion without the s and you get 21 hits.
Whoa thanks for posting this. Bookmarked. This lends me to believe JohnHereToHelp on this subject. I was beginning to grow suspicious of him but maybe he is really telling the truth.
It's not a prion or even prion-like, that word is thrown in there for impact potential.