In the heart, myocarditis can induce cell loss, interstitial and replacement fibrosis, wall motion abnormalities, decreased ejection fraction, and arrhythmias. Moreover, myocarditis is one of the leading causes of Sudden cardiac death
The cause of myocarditis can among others be an allergic or toxic reaction to medicines and toxic drugs as well as autoimmune organ-specific myocarditis and systemic autoimmune diseases-associated myocarditis. However, most often, the cause of myocarditis is an infection, including viruses, bacteria, protozoa, and fungi.
In our case, the cause is an TRANSFECTION with the Spike Protein of SARS-CoV-2. The infection of the cardiac endothelium can cause among others endothelial activation, damage, and permeability. For instance, infection of cardiac endothelial cells in patients with viral myocarditis has shown to induce endothelial microparticles reflecting endothelial damage.
What is the discussion? Whether this or that?
Or are we discussing how a and B and C can come together and cause certain observed phenomena.
Because these structures functioning as large clots, have not even been recognized by " science" .