Could you provide the exact product you are interested in? I can call the lab that manufacturers it on Monday (labs normally don't work on weekends) and I can report back to you if they are on the up and up.

Here is some further reading for those interested. Feel free to ask questions.

Mechanism of Action:

1. Inhibition of Lipoprotein(a) Binding

• Lipoprotein(a) [Lp(a)] and Atherosclerosis: Lp(a) is structurally similar to low-density lipoprotein (LDL) but contains an additional protein called apolipoprotein(a), which has a high affinity for damaged arterial walls. It accumulates at sites of endothelial injury, contributing to the development of atherosclerotic plaques.
• Lysine's Role: Lysine competes with Lp(a) for binding to lysine-binding sites present on the arterial wall. By occupying these binding sites, lysine prevents Lp(a) from adhering to the vascular endothelium, reducing the formation and progression of calcified plaques.
• Collagen Stabilization: Lp(a) also binds to fibrin and damaged collagen within atherosclerotic lesions, further promoting calcification. Lysine, by blocking Lp(a) and stabilizing collagen structures, helps prevent further deposition of lipoproteins and calcium.

2. Solubilization of Lipoprotein(a)

• Lysine’s Chelating Action: Lysine, along with other amino acids like proline, can enhance the solubility of Lp(a) already bound to arterial walls, helping to mobilize and dissolve existing calcified plaques. This occurs because lysine disrupts the electrostatic interactions between Lp(a) and the arterial matrix, allowing the plaque to be metabolized and cleared from the bloodstream.
• Calcium Dissolution: Lysine’s chelating properties may also contribute to the dissolution of calcium phosphate deposits in the arterial wall. Though this mechanism is less well-characterized, it is postulated that lysine interacts with calcium ions and prevents their crystallization, thereby reducing calcification.

3. Reduction of Inflammation and Oxidative Stress

• Inflammation in Atherosclerosis: Inflammatory processes within the vasculature attract immune cells that produce reactive oxygen species (ROS) and cytokines, which promote calcification by damaging vascular smooth muscle cells (VSMCs) and endothelial cells.
• Lysine's Anti-inflammatory Role: Lysine has shown some capacity to modulate inflammatory responses. By reducing oxidative stress and inflammatory mediators, lysine may indirectly prevent the calcification process, as oxidative stress accelerates VSMC calcification.

4. Interaction with Matrix Gla Protein (MGP)

• MGP and Calcification: MGP is an important inhibitor of vascular calcification. Its function is dependent on vitamin K, but some evidence suggests that lysine can also influence MGP activity, helping to prevent calcification.
• Lysine and Calcium Metabolism: Lysine may enhance calcium metabolism by interacting with calcium-binding proteins or promoting the activity of inhibitors like MGP, thereby reducing calcification by limiting the deposition of calcium salts in arterial tissue.

5. Enhancement of Collagen Production and Vascular Repair

• Collagen Synthesis: Lysine is critical for collagen production, a major structural component of the extracellular matrix in arteries. By supporting collagen formation, lysine helps maintain the integrity of the arterial wall, which may be important in preventing the mechanical damage that triggers Lp(a) and calcium deposition.
• Vascular Repair: Enhanced collagen production promotes the repair of damaged arterial walls, reducing the binding sites for Lp(a) and preventing subsequent calcification.

6. Synergistic Role with Vitamin C

• Vitamin C and Collagen Stabilization: Lysine often works in tandem with vitamin C to promote collagen synthesis and stabilize vascular walls. Vitamin C is essential for hydroxylation of proline and lysine residues in collagen, which strengthens the arterial matrix. This reduces the availability of exposed collagen and fibrin, both of which can serve as substrates for Lp(a) and calcified plaques.
• Antioxidant Synergy: Lysine and vitamin C may synergistically reduce oxidative stress, further limiting vascular calcification and plaque progression.

*** Mechanical Rational for Timing***

1. Avoiding Protein with Lysine:

• Competition with Other Amino Acids: Lysine, like other amino acids, is absorbed in the small intestine via specific transporters. If lysine is consumed along with large amounts of protein, the presence of other amino acids may compete for the same transporters, potentially reducing the absorption efficiency of lysine. This could limit the amount of lysine available for its proposed beneficial effects on vascular health, such as preventing Lp(a) binding and supporting collagen synthesis.
• Metabolic Load: High protein intake increases the metabolic load on the liver and kidneys due to the need to metabolize and excrete excess nitrogen (in the form of urea). This could divert metabolic resources from the synthesis and repair processes that lysine is involved in, such as collagen production and endothelial repair. For those focused on optimizing lysine’s vascular benefits, it may be more effective to take lysine separately, especially if the goal is to avoid competition or interference from other amino acids.

2. Avoiding Carbohydrates with Vitamin C:

• Glucose-Vitamin C Competition: Glucose and vitamin C share similar transport pathways, particularly the GLUT1 and GLUT3 transporters, which are responsible for transporting both substances into cells. High blood sugar levels or excessive carbohydrate intake can reduce the efficiency of vitamin C uptake by endothelial cells, as glucose competes with vitamin C for transport into the cells. This competition can result in reduced intracellular concentrations of vitamin C, impairing its antioxidant and endothelial-protective functions, such as nitric oxide bioavailability and collagen synthesis.
• Glycation and Oxidative Stress: High carbohydrate intake, especially refined sugars, can lead to the formation of advanced glycation end-products (AGEs). AGEs are harmful compounds that form when proteins or fats combine with sugars in the bloodstream. AGEs promote oxidative stress, inflammation, and endothelial dysfunction. Vitamin C, which is critical for reducing oxidative stress and preventing endothelial damage, may be less effective if consumed alongside high levels of carbohydrates that increase glycation processes.
• Hyperglycemia-Induced Endothelial Dysfunction: High carbohydrate intake, particularly in individuals with insulin resistance or poor glucose control, can lead to postprandial (after-meal) hyperglycemia, which is known to cause endothelial dysfunction. Vitamin C's ability to protect endothelial cells from damage could be compromised when paired with excessive carbohydrates, as the oxidative stress and inflammation from hyperglycemia may outweigh the protective effects of vitamin C.

HOPE THAT HELPS

Great read and great information from a fellow researcher!! The pharmacokinetic and pharmacodynamic profiles of both lysine and vitamin C exhibit high bioavailability under typical conditions. While liposomal formulations offer an enhancement in bioavailability, such modifications are generally unnecessary given the sufficient absorption rates of these compounds. But if you can afford it, go for it. You stated that the brand doesn't matter but it does. Some places can mix up to 40% with other compounds. So if the brand you stated worked for you, keep it! I typically call the manufacturer and have them send over their lab data, clinical data, etc.

If you're taking fenbendazole and ivermectin together, it's important to monitor for any signs of liver toxicity, such as fatigue, jaundice, or dark urine, as both medications are metabolized by the liver. Stay hydrated by drinking plenty of water, which can help support your liver and overall health.

You are correct that most cancers exhibit the Warburg effect, where glycolysis occurs even in the presence of oxygen. Fenbendazole directly inhibits glycolysis, while compounds such as curcumin, resveratrol, quercetin, and EGCG indirectly suppress this metabolic pathway. Reducing carbohydrate intake would be beneficial, though it is not strictly necessary if these agents are being taken, as they provide metabolic modulation to counteract the elevated glycolysis.

Hi. I will provide indepth information on what you should be taking. I need to know what cancer, what stage, do you know your PDL1 level, have you done any genetic testing?

• PLEASE COMMENT BACK SO I CAN HELP YOU WITH CLINICAL INFORMATION

edit for quick info

• Fenbendazole 888 mg 3 times a week. https://fenben.pro/product/fenbendazol-capsules-444-mg-30-units/
• Nano-Curcumin 3 times a day https://a.co/d/ir48kpZ
• Nano-Resveratrol 3 times a day https://a.co/d/hAQpXyf
• Liposimal-EGCG 1 time in the morning https://a.co/d/1flyqrH
• Nano Quercetin 1 time a day https://a.co/d/eS5KAO5
• ARMRA Colostrum 1 time in the morning https://a.co/d/gO9yxQz
• Zink

Please let me know if you want clinical rational for each

Me too, me too

Yes bpc157! Good job on the suggestion!

• If you are looking for anti-inflammatory effects I would suggest Nano-curcumin specifically this one: https://a.co/d/2mKmdQU Remove it from its shell directly into your mouth (tastes good)

• Another would be: Body Protective Compound 157 https://a.co/d/1UPmC5G

Let me know and I will write up a clinical review on them for you

Hello, I research pharmacokinetics and pharmcodynamics of various phytochemicals, drugs, etc. What is your goal? And what info would you like on carbon60

edit after some preliminary research

• Mechanism of Action:

  • Acts as a powerful antioxidant, scavenging free radicals and reducing oxidative stress.
  • May reduce inflammation by downregulating pro-inflammatory cytokines.
  • Demonstrates potential neuroprotective effects, crossing the blood-brain barrier.

• Pharmacokinetics:

  • Enhanced bioavailability when dissolved in oils (e.g., olive, coconut, avocado oil).
  • Absorbed in the gastrointestinal tract; distributed throughout the body.
  • Primarily excreted through fecal matter, with limited bioaccumulation.
  • Long half-life and stable structure suggest prolonged biological activity.

• Safety Profile:

  • Low acute toxicity in preclinical studies, especially in oil-based forms.
  • No significant hepatotoxicity, nephrotoxicity, or blood alterations in animal models.
  • Minimizes phototoxicity risk when stored away from UV light.

• Potential Benefits:

  • Antioxidant: Reduces oxidative damage to cells, proteins, and DNA.
  • Anti-inflammatory: May alleviate chronic inflammation in conditions like arthritis and cardiovascular disease.
  • Neuroprotection: Potential protection against neurodegenerative diseases (e.g., Alzheimer's, Parkinson's).
  • Anti-aging: Hypothesized to reduce cellular aging through free radical scavenging.

• Clinical Use Considerations:

  • Oil-based formulations recommended for enhanced absorption; olive oil-based C60 is the most studied.
  • May be beneficial for patients with oxidative stress or inflammatory conditions.
  • Caution advised in patients with pre-existing liver or kidney conditions.
  • Monitor for interactions with other pharmaceutical agents and potential phototoxicity.

I did read what you said, hence why I am asking you to provide greater detail because I am interested.

Did anybody read the part where the 2020 election HAS to be overturned before November 5? Military and SCOTUS?

• No, where can I read it. Elaborate.
• Is this verifiable at all or just something fun to think about?

Elaborate

• I personally know nothing about Dave Dave.
• Those on here that know me know I am 100% against the idea Biden is an actor in a mask. An I get a lot of flack for it but stand by it. HOWEVER
• The second video, I don't know where it's from or who it is, all I can say is that's an actor in a mask

NO. this was a celebration. People were lighting fire words over by me as well.

Replacement psyop is just that. A psyop just like flat earth A reptilian overlords. It's designed to make us look ridiculous. Stop falling for it.

The most bioavailable form of collagen is hydrolyzed collagen, also known as collagen peptides. BPC-157 Body Protection Compound-157 is another peptide that would provide a more clinical difference though or taken alonside collegen peptides. These both have roughly 95% bioavailability.

Collagen's Clinical Mechanism for Joint Healing

1. Cartilage Repair and Maintenance

   • Mechanism: Collagen, particularly type II collagen, is the primary protein in articular cartilage, which cushions joints. When collagen is degraded (e.g., due to injury or osteoarthritis), cartilage loses its integrity. Supplementing collagen peptides helps provide the building blocks for synthesizing new collagen fibers in the extracellular matrix, supporting cartilage repair.
   • Clinical Impact: Collagen peptides stimulate chondrocytes (cells that produce cartilage) to synthesize more collagen and proteoglycans, improving the structural integrity of the cartilage and potentially reducing joint pain.

2. Reduction of Inflammation

   • Mechanism: Collagen peptides may have anti-inflammatory effects by modulating immune cell activity in the joint space. This can help reduce joint swelling and prevent further damage.
   • Clinical Impact: By lowering inflammation, collagen supplementation helps create a more favorable environment for tissue repair and healing.

3. Enhanced Tissue Regeneration

   • Mechanism: Collagen promotes fibroblast proliferation, which leads to the production of connective tissue that supports tendons and ligaments. These tissues are vital for joint stability and movement.
   • Clinical Impact: Collagen peptides improve the strength and elasticity of tendons and ligaments, reducing injury risk and promoting faster recovery after joint trauma.

4. Increased Synthesis of Extracellular Matrix Components

   • Mechanism: Collagen peptides stimulate the synthesis of glycosaminoglycans (e.g., hyaluronic acid) and elastin, which are essential for maintaining joint lubrication and resilience.
   • Clinical Impact: This contributes to the overall health and function of synovial joints by improving flexibility and reducing friction between articulating surfaces.

BPC-157's Mechanism and Why It May Be Useful for Joint Healing

BPC-157 is a peptide with powerful regenerative and healing properties, and its potential use in joint healing is supported by several mechanisms:

1. Angiogenesis (Formation of New Blood Vessels)

   • Mechanism: BPC-157 promotes angiogenesis, improving blood supply to injured areas, including joints. This enhanced blood flow delivers oxygen and essential nutrients, accelerating tissue repair.
   • Clinical Impact: Joint tissues like tendons and ligaments often have poor blood supply, which delays healing. BPC-157's ability to stimulate angiogenesis makes it particularly useful for repairing these areas.

2. Accelerated Collagen Synthesis

   • Mechanism: BPC-157 stimulates fibroblasts to produce more collagen, which is crucial for the repair and regeneration of connective tissues such as tendons, ligaments, and cartilage.
   • Clinical Impact: By boosting collagen production, BPC-157 can promote faster healing of joint-related tissues, restoring their structural integrity and function.

3. Anti-Inflammatory Effects

   • Mechanism: BPC-157 has been shown to reduce the production of pro-inflammatory cytokines, decreasing inflammation in injured tissues. It also helps protect tissues from oxidative stress, further limiting inflammation and damage.
   • Clinical Impact: Reducing inflammation not only relieves pain but also prevents further tissue degradation, creating a better environment for joint repair.

4. Protection and Healing of Tendons and Ligaments

   • Mechanism: BPC-157 enhances the healing of tendons and ligaments by modulating various growth factors and promoting cell proliferation and migration.
   • Clinical Impact: Tendon and ligament injuries, common in joint trauma, can benefit from the peptide's ability to stimulate repair and reinforce the strength of these structures.

5. Joint Capsule Integrity and Synovial Fluid Production

   • Mechanism: BPC-157 may protect the joint capsule (the fibrous tissue surrounding joints) and stimulate the production of synovial fluid, which lubricates and nourishes the joint.
   • Clinical Impact: Improved joint lubrication can reduce friction and wear on the cartilage, slowing the progression of joint degeneration and improving overall joint function.

If I were her I would take the following:

• Hydrolyzed Collagen Powder: https://a.co/d/7GuWKty

• BPC 157 https://a.co/d/2kShpXk

• Nano- Curcumin https://a.co/d/3uYE7Pw

I would reccomend just a lot of study. I got into it due to my dad getting stage 4 lung cancer. So I dived deep into research.

Word play? What can you specifically articulate that I said which would constitute wordplay?

• This is psywar. Trust nothing you cannot verify yourself. That goes for information from either side. That I would agree with you.

Masks are real and can be very convencing. But there is not a single mask that adheres to all of the following at once:

• Looks like another known individual. (Non-generic)
• Appears hyper-realistic
• Flexible with fine wrinkles and facial expressions.
• matches facial wrinkle mapping of the target.

forensic evidence of someone wearing a mask without physically contacting the peraon would include but is not limited to:

• forensic facial wrinkle mapping
• forensic audio analysis

If you are insinuating that it's a mask,

• i did not assume, hance the preface with this first sentence.

Not exactly. It's the positive claim the requires evidence. But nonetheless I have done my due diligence, and did a forensic facial wrinkle mapping both of Biden and one of the people that are claimed to be JFK Jr. Biden matches the old one. Surprisingly, there is a person matching JFK Jr's facial wrinkle mapping. The image is not very clear so the quality is a little low but it's Surprisingly similar.

Yes, but don't forget to remain skeptical.

Exactly. It's a flat earth, reptilian overlord, type psyop designed to distract and discredit the movement. Unfortunately the majority has fallen for it this time.

Simple. If you can provide forensic evidence they are masked, show the evidence. If not, keep an open mind but don't assume something without the hard evidence.

For the one who downvoted this; question own cognitive dissonance, what exactly did I say above is worth down voting?

Yep.