Thanks for that, and as somebody who has had 3 blood clots, one was PE I am well aware they happen. Mine happened through paralysis due to a complete burst fracture of my L2 vertebrae. Yes Eliquis is the current blood thinner that holds the required INR level.
But when you look at the statistics, just like the other common adverse reactions, blood clots have climbed to huge levels AFTER injection.
Was the blood clot from the incessant coughing which caused a bleed?
The reason for the clot development is that the virus attacks the heme in the blood cells, rendering them unable to carry the required oxygen. The lungs become distressed and become inflamed because they think they are falling down on the job and start working harder, but it doesn't help. This will trigger the cough reflex.
Additionally, the vascular system realizes something is wrong because not enough oxygen is being delivered. Its response is to assume the body is wounded and has a bleed. So, it makes more red blood cells and sends out clotting factor (fibrin) to plug the leak, wherever it is. But there is no wound, there is no leak. All these extra red blood cells get attacked by the virus and they, too cannot carry enough oxygen.
So, the combination of too many damaged blood cells packed in the veins along with too much fibrin starts to create these tiny micro clots (microthrombosis). They won't show up on an x-ray or CT scan until they get big. But enough of them will do you in.
Currently, the only thing that will confirm their development is the D-Dimer test. The higher the number, the more clots you have.
Thus, the cough won't go away until the lungs are allowed to relax. The lungs won't relax until the blood starts carrying more oxygen. Some relief can be gained with supplemental oxygen coupled with the steroids.
The solution was to try and stop the virus with Ivermectin + vitamins, and assist the body with oxygen, antibiotics, and anti-coagulants until his body could get a handle on the situation.
With all due respect to u/casuallyobservant, there is an additional and possibly more direct explanation for the clotting. The spike protein seeks cells with the ACE-2 receptor to infect. Those cells, found throughout the body, are primarily in endothelial cells, which are the cells that line the interior of the blood vessels, i.e. the veins and arteries. When these cells are damaged it usually signifies a broken blood vessel that requires clotting in order to stop a person from losing blood; in the case of Covid the cells are damaged from within, which still triggers the clotting but now the clotting factor is released within the blood stream rather than forming a scab outside the blood vessel. Worse, the clot-attracting blood is now circulating along with the virus to potentially lodge anywhere throughout the circulatory system. I've seen this explained in a number of places, but medcram.org has some of the best explanations.
It also affects heme directly, which is why different blood types are affected differently (apparently O is slightly better), but I know hemotologists who don't agree, and I haven't seen agreement on this, while the endothelial damage is much more evident. For what its worth.
You are correct. I failed to mention the damage to the endothelium that also occurs. Those videos I've seen where the microscopic examination of the blood cells shows such horrible damage to the cells themselves is appalling. Then, coupled with the damage to the lining in all of the pathways that carry the blood is a tragedy.
Thanks for clarifying further what I was attempting to explain.
Thanks for that, and as somebody who has had 3 blood clots, one was PE I am well aware they happen. Mine happened through paralysis due to a complete burst fracture of my L2 vertebrae. Yes Eliquis is the current blood thinner that holds the required INR level.
But when you look at the statistics, just like the other common adverse reactions, blood clots have climbed to huge levels AFTER injection.
Was the blood clot from the incessant coughing which caused a bleed?
This explanation should help:
The reason for the clot development is that the virus attacks the heme in the blood cells, rendering them unable to carry the required oxygen. The lungs become distressed and become inflamed because they think they are falling down on the job and start working harder, but it doesn't help. This will trigger the cough reflex.
Additionally, the vascular system realizes something is wrong because not enough oxygen is being delivered. Its response is to assume the body is wounded and has a bleed. So, it makes more red blood cells and sends out clotting factor (fibrin) to plug the leak, wherever it is. But there is no wound, there is no leak. All these extra red blood cells get attacked by the virus and they, too cannot carry enough oxygen.
So, the combination of too many damaged blood cells packed in the veins along with too much fibrin starts to create these tiny micro clots (microthrombosis). They won't show up on an x-ray or CT scan until they get big. But enough of them will do you in.
Currently, the only thing that will confirm their development is the D-Dimer test. The higher the number, the more clots you have.
Thus, the cough won't go away until the lungs are allowed to relax. The lungs won't relax until the blood starts carrying more oxygen. Some relief can be gained with supplemental oxygen coupled with the steroids.
The solution was to try and stop the virus with Ivermectin + vitamins, and assist the body with oxygen, antibiotics, and anti-coagulants until his body could get a handle on the situation.
Yes thanks, it's helped a lot.
With all due respect to u/casuallyobservant, there is an additional and possibly more direct explanation for the clotting. The spike protein seeks cells with the ACE-2 receptor to infect. Those cells, found throughout the body, are primarily in endothelial cells, which are the cells that line the interior of the blood vessels, i.e. the veins and arteries. When these cells are damaged it usually signifies a broken blood vessel that requires clotting in order to stop a person from losing blood; in the case of Covid the cells are damaged from within, which still triggers the clotting but now the clotting factor is released within the blood stream rather than forming a scab outside the blood vessel. Worse, the clot-attracting blood is now circulating along with the virus to potentially lodge anywhere throughout the circulatory system. I've seen this explained in a number of places, but medcram.org has some of the best explanations.
It also affects heme directly, which is why different blood types are affected differently (apparently O is slightly better), but I know hemotologists who don't agree, and I haven't seen agreement on this, while the endothelial damage is much more evident. For what its worth.
Thanks for the post.
You are correct. I failed to mention the damage to the endothelium that also occurs. Those videos I've seen where the microscopic examination of the blood cells shows such horrible damage to the cells themselves is appalling. Then, coupled with the damage to the lining in all of the pathways that carry the blood is a tragedy.
Thanks for clarifying further what I was attempting to explain.