"Deez Nutz"?
by
Unfortunately I looked further into this video, and its a fake. the structure shown in the video is nothing other than sodium chloride (salt) Here is simple examples: https://m.media-amazon.com/images/I/41PXqhBkltL.jpg
Someone was saying how Trump cheated, and they need to do a full national recount.
I simply said: We claimed there was cheating in 2020, now you claim there was cheating this time around. Maybe now, election integrity & security could be considered bipartisan.
Apparently, that's too much logic for libs to handle on there.
Simple: make a deal with her neither of you will try and challenge or claim Trump will do or say or has said anything untill you both watch his speech or whatever in totality. And you will both try to watch his speeches and talk about what you agree with and disagree with. Do not Bing his past or what other media is saying about it. You must both use internal critiques.
Either you will learn to be more centered or she will. Let's wait and see. Use truth and reality as the measure not what some form of media tells you.
Prayer
Our father in heaven, all glory be to you. We pray you put these people into your hands and deliver to them your will. We lift our fears and burdens and give them to you. We love you, and we trust you. Amen
Quick List
- Nanocurcumin
- Liposomal EGCG
- Nano resveratrol
- Lysine
- Vitamin C
- Alpha Lipoic Acid
- BPC-175
- Nattokinase
- Ivermectin
Clinical Reasoning
Nanocurcumin
- Anti-inflammatory: Nanocurcumin, a bioavailable form of curcumin, inhibits NF-kB, a transcription factor that regulates inflammation. By downregulating pro-inflammatory cytokines (e.g., TNF-α, IL-6), it may reduce inflammatory responses linked to myocarditis and pericarditis.
- Antioxidant: Curcumin’s antioxidant properties help to mitigate oxidative stress and ROS production, potentially counteracting the oxidative damage induced by spike proteins.
- Cardioprotective: Curcumin has demonstrated cardioprotective effects by reducing lipid peroxidation and protecting myocardial tissue against inflammation and fibrosis.
Liposomal EGCG (Epigallocatechin Gallate)
- Antioxidant: Liposomal EGCG, the active catechin in green tea, supports cellular defense against oxidative stress. It reduces ROS levels, which are implicated in cellular damage associated with spike protein toxicity.
- Anti-inflammatory: EGCG modulates cytokine production, decreasing IL-6 and TNF-α, which are associated with inflammatory heart conditions like myocarditis.
- Inhibition of Spike Protein Activity: EGCG has shown potential in inhibiting spike protein’s interaction with ACE2 receptors, potentially reducing spike protein-mediated cellular damage.
Nano Resveratrol
- Anti-inflammatory and Antioxidant: Nano-resveratrol exhibits enhanced bioavailability, allowing it to more effectively combat inflammation and oxidative stress, reducing IL-1β, IL-6, and TNF-α.
- Mitochondrial Protection: Resveratrol helps in maintaining mitochondrial function and reducing apoptosis in cells exposed to spike protein-induced stress.
- Cardioprotective: Resveratrol has been shown to improve endothelial function and reduce fibrosis, helping mitigate myocardial damage in cases of myocarditis.
Lysine
- Anti-inflammatory: Lysine helps in modulating immune responses, potentially reducing inflammation associated with pericarditis and myocarditis.
- Arterial Plaque Reduction and Healing: Lysine has been associated with preventing arterial plaque buildup and supporting vascular healing, which may help protect against cardiovascular issues related to inflammatory heart conditions.
- Spike Protein Interaction: Lysine may interfere with the attachment of the spike protein to host cells, possibly mitigating cellular damage linked to spike protein interactions.
Vitamin C
- Antioxidant: High-dose vitamin C combats oxidative stress and neutralizes free radicals, helping to protect myocardial cells from oxidative damage associated with inflammation and immune responses.
- Anti-inflammatory: Vitamin C reduces pro-inflammatory cytokine levels, which may support reduced inflammation in myocarditis and pericarditis cases.
- Arterial Plaque Reduction and Injury Healing: Vitamin C plays a role in collagen synthesis, contributing to vascular healing and possibly reducing plaque buildup, providing support in cardiovascular repair and resilience.
- Immune Support: Vitamin C enhances immune cell function, aiding in tissue repair and recovery following immune-mediated damage.
Alpha Lipoic Acid (ALA)
- Antioxidant: ALA is a potent antioxidant that scavenges ROS, supports mitochondrial function, and mitigates spike protein-induced cellular damage.
- Anti-inflammatory: ALA reduces the activation of NF-kB and downregulates inflammatory markers, potentially helping to reduce inflammation in cardiac tissues.
- Cardioprotective: ALA has demonstrated protective effects on vascular endothelium and may prevent oxidative damage to cardiac cells.
BPC-157 (Body Protection Compound-157)
- Anti-inflammatory: BPC-157 modulates inflammatory pathways and has been shown to accelerate healing of damaged tissues, particularly in cardiovascular injury.
- Tissue Repair: BPC-157 promotes cellular regeneration and enhances angiogenesis, potentially aiding in recovery of cardiac tissue affected by myocarditis or pericarditis.
- Protective Against Oxidative Stress: BPC-157 has shown properties that may protect against oxidative damage in injured tissues, supporting overall cellular health.
Nattokinase
- Fibrinolytic Enzyme: Nattokinase dissolves fibrin, potentially reducing clotting risks associated with spike protein interaction in the vascular system.
- Spike Protein Degradation: Nattokinase has shown potential in degrading spike protein in laboratory studies, which may mitigate spike protein-induced damage to endothelial cells.
- Cardiovascular Support: Nattokinase enhances blood flow and may reduce hypertension and arterial stiffness, supporting heart health in patients with inflammatory heart conditions.
Ivermectin
- Anti-inflammatory: Ivermectin downregulates cytokines such as IL-1 and IL-6, reducing systemic and localized inflammation, which may benefit patients with myocarditis and pericarditis.
- Spike Protein Binding Inhibition: Ivermectin has shown affinity for spike protein binding sites, potentially preventing its interaction with ACE2 receptors and reducing cellular damage.
- Antioxidant Properties: By reducing mitochondrial oxidative stress, ivermectin may help prevent apoptosis in cells exposed to spike protein toxicity.
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He can't.