This isnt a video by the harris campaign its made by a random person showing you how they could have done it.

Some dude made this using ai, the ai likely didnt understand that this was stairs and not an escalator.

• TLDR: No, it will help fight the cancer while allowing chemo to do its job.

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• Each of these treatment approaches targets cancer cells through different mechanisms:

• Chemotherapy primarily disrupts DNA processes

• Fenbendazole may interfere with cancer cell metabolism and support immune function by reducing the cancer cells ability to remove unwanted substances.

• Targeted therapies and immunotherapies focus on specific cancer cell characteristics or enhancing the immune response, respectively.

• Phytochemicals can have various effects, including influencing drug resistance mechanisms and signaling pathways. Combining these strategies can potentially improve treatment outcomes.

https://fenben.pro/

Likely, what happening is they are inputting a real video with a limited set of real people. The ai fills in the resr of the people.

Doesn't look like a nuke

interpretation

• This is a map of the intracellular pathways pathways of cancer. In this case I was focusing in lung cancer but many cancers share similar pathways.
• Any black box is a "start point" of a pathway. Pathways can have more than one start.
• the colored boxes are drugs and they point to what step in the process it inhibits.
• there is a key on the side.

Creation

• This was created with an online program called miro.
• I first mapped just the cancer pathways
• I then looked up phytochemicals, and other drugs which inhibit specific pathways and their mechanism.

meaning

• it's to help communicate how to fight cancer.

There is no need to greet it. Chat gpt is not a sentient being.

Where is the garden hose getting water?

Main issue to me would be water.

Not only that, it also inhibits p-gp. P-gp is overexpressed in tumor cells, contributing to multidrug resistance (MDR). By pumping drugs out of cancer cells, P-gp can reduce the drugs' effectiveness, making the cancer harder to treat. Check out this diagram I created. Fenbendazole is near the bottom. https://i.ibb.co/5M43BP0/Lung-Cancer.png

I created this to help: https://i.ibb.co/5M43BP0/Lung-Cancer.png

Just FYI, if you plan to add phytochemicals to your treatment purchase nano, or liposomal versions. For example: curcumin has roughly 1% absorption. Add peperine and it goes up to 5% up to 20%. In nano form that can get to 40% up to 90+%

Whats unique about fenbendazole, ivermectin etc. Other than high bioavailability Is that it inhibits p-gp pumps, cells use this to push unwanted stuff out which limits the effect of tcells, and drugs. Turning it off is like removing one of its shields.

If anyone wanna know exactly why this works I can give you details

Thanks for your fight, enjoy your rest

Sounds like you have it under control 👍

You are very welcome, any time if you ever have clinical questions on desease or suppliments let me know and I will gladly give you what I know, and do some research for you as well.

If you are set on speeding it up, Oxytocin Is what the vet would use. https://www.amazon.com/Revival-Animal-Health-Breeders-Supplement/dp/B00JHYFKE6/ref=sr_1_1?crid=1KR23AGOJ153R&dib=eyJ2IjoiMSJ9.pPv8iZNMUxqMl7bGEI0IWncpp7gK6vVP3fLOd9_-lUOqB99hHi0to_8PrWBLiKnxZcuLClLXsqW3FrLyqrPzuGROXmyLpO1hI2_q_OHbZ672lq15CFpT1Qwu4Dy2gIWJtR6kDmJAleeczV_fGbKDEIL3Xmj0tVJt0IK7NNwzoE05Wh7SJLNLKkjEUGCfdObi_1idirdY-coe6TRCU60h6SkinnCU8mSRMjbfH9RC_i8.4QLjUBtKOwMiPbS3ZZ_2lz6SzaTpE4Sba_7lg1FxIdk&dib_tag=se&keywords=oxytocin+for+dogs+in+labor&qid=1723260697&sprefix=Oxytocin+lab%2Caps%2C97&sr=8-1

• Copy, what I wrote into any search engine you want, you won’t find it. Instead look at my history and you will see, when it comes to medical questions I go out and research to help others.

• now, for the sake of argument what specifically is false? The common bacteria associated? The common medications used? The palliative care? The phytochemicals? Nano phytochemicals having higher bioavailability? What.

People that moved away from Jesus and became secular atheists/agnostic removed their belief in God but that doesn’t remove their religiosity. They instead created their own dogma, and their own priests.

Why are you claiming that I am simply “copying information from the net” actually read what was written fully. Maybe your cellulitis wouldn’t have been as bad if you followed it. But now that you are here, instead of disparaging my research and my help, you could share what relieved your issues if you found any. If you think a thing I wrote is nonsense then address that specifically.

It definitely could. I am also always willing to dive deep into health research if anyone has specific questions.

Think of it this way: we know him. We know what he is about. He doesn't need to market to us anymore. He is trying to capture those in the fence.

As much as i enjoy your research and enthusiasm, i would like to add info to help others.

• phytochemicals have very low bioavailability. For example, roughly 1% of injested curcumin is absorbed into cells. Adding peperine can boost absorption to 5% up to 20% but like many phytochemicals, using nanocurcumin or liposomal curcumin is much much higher bioavailability roughly 90% So if anyone wants to take phytochemicals, make sure to use ones that are bioavailable.

Here is some research I went ahead and did. It's the mechanism of Action of cellulitis, it's damage, risks and ways to treat it.

Mechanism of damage

• 1. Bacterial Invasion:

  • Entry and Spread: Bacteria, usually Staphylococcus aureus or Streptococcus pyogenes, enter through a break in the skin. Once inside, they begin to multiply and spread through the skin’s deeper layers, including the dermis and subcutaneous fat.

• 2. Immune Response:

  • Inflammatory Response: The body's immune system responds to the infection by sending white blood cells (such as neutrophils) and inflammatory mediators (like cytokines) to the site of infection.
  • Vasodilation and Increased Permeability: Inflammatory mediators cause blood vessels to dilate and become more permeable, leading to increased blood flow and leakage of fluid into the tissues. This results in swelling (edema).

• 3. Tissue Damage:

  • Inflammation: The inflammatory response, while necessary to fight the infection, also causes collateral damage to surrounding tissues. The accumulation of immune cells and inflammatory mediators can lead to tissue damage.
  • Enzymes and Toxins: Bacteria produce enzymes and toxins that contribute to tissue breakdown. These substances can further damage skin cells and extracellular matrix components.

• 4. Formation of Abscesses:

  • Pus Formation: As the immune system tries to contain the infection, pus (a collection of dead white blood cells, bacteria, and tissue debris) can form. In severe cases, this pus can accumulate and create an abscess.

• 5. Systemic Effects:

  • Sepsis Risk: If the infection spreads to the bloodstream (bacteremia), it can lead to sepsis, a life-threatening condition characterized by widespread inflammation and organ dysfunction.

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Treatment of Cellulitis

1. Direct Methods to Treat Cellulitis:

1.1. Antibiotic Therapy

• Objective: To eliminate the bacterial infection causing cellulitis.
• Common Antibiotics:
  • Penicillin: First-line treatment for Streptococcus pyogenes infections.
  • Amoxicillin: Alternative to penicillin, effective against a broad range of bacteria including Streptococcus and Staphylococcus species.
  • Cephalosporins: Used for patients with penicillin allergies or in more severe cases.
  • Erythromycin/Azithromycin: Macrolides used for penicillin-allergic patients or in cases of resistance.
  • Clindamycin: Effective for severe infections and known for activity against Staphylococcus aureus, including MRSA.

Reasoning: Antibiotics target and eliminate the bacterial pathogens responsible for cellulitis, reducing infection and preventing complications such as abscess formation and sepsis.

1.2. Wound Care

• Objective: To prevent further bacterial entry and manage infected areas.
• Protocols:
  • Cleaning: Thoroughly clean the affected area with mild soap and water.
  • Dressing: Use sterile dressings to cover wounds and prevent contamination.
  • Monitoring: Regularly inspect the wound for signs of worsening infection or complications.

Reasoning: Proper wound care helps prevent additional bacterial entry and reduces the risk of spreading the infection.

1.3. Supportive Measures

• Objective: To manage symptoms and support the body’s healing process.
• Protocols:
  • Elevation: Elevate the affected limb to reduce swelling.
  • Rest: Encourage rest to support the immune system.
  • Pain Management: Use nonsteroidal anti-inflammatory drugs (NSAIDs) or acetaminophen for pain and inflammation.

Reasoning: These measures help alleviate symptoms such as pain and swelling, supporting the body’s overall recovery.

2. Phytochemicals to Treat Symptoms and Prevent Sepsis:

2.1. Garlic (Allicin)

• Objective: To provide additional antimicrobial support.
• Mechanism:
  • Antibacterial Activity: Allicin inhibits bacterial enzymes and disrupts bacterial cell wall synthesis, reducing bacterial growth.
  • Immune Support: Allicin enhances white blood cell activity.
• stabilized Version: Stable-allicin can improve bioavailability and stability.
  • https://a.co/d/95bjjNW
  • Bioavailability Change: Nano-allicin formulations can increase absorption and efficacy compared to standard allicin by up to 10 times.

Reasoning: Nano-allicin offers enhanced stability and bioavailability, potentially providing more effective antimicrobial and immune-supportive benefits.

2.2. Turmeric (Curcumin)

• Objective: To reduce inflammation and support immune function.
• Mechanism:
  • Anti-inflammatory: Curcumin inhibits inflammatory cytokines and enzymes, reducing inflammation.
  • Antioxidant: Curcumin neutralizes free radicals and reduces oxidative stress.
• Nano Version: Nano-curcumin improves solubility and absorption.
  • https://a.co/d/akqqbiJ
  • Bioavailability Change: Nano-curcumin can increase bioavailability by up to 30 times compared to conventional curcumin.

Reasoning: Nano-curcumin’s enhanced absorption and bioavailability help maximize its anti-inflammatory and antioxidant effects, supporting better management of cellulitis symptoms.

2.3. Ginger (Gingerol)

• Objective: To reduce inflammation and provide additional antimicrobial support.
• Mechanism:
  • Anti-inflammatory: Gingerol reduces pro-inflammatory cytokines and inhibits inflammatory pathways.
  • Antimicrobial: Gingerol has direct antibacterial properties.
• Nano Version: Nano-gingerol enhances bioavailability and stability.
  • https://a.co/d/aTIWbkr
  • Bioavailability Change: Nano-gingerol can improve absorption by up to 15 times compared to standard gingerol.

Reasoning: Nano-gingerol’s improved absorption and stability can enhance its anti-inflammatory and antimicrobial effects, providing better support for managing cellulitis.

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Sources

• https://doi.org/10.1016/j.jsps.2022.09.015
• https://doi.org/10.3390/ijms25073617

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Lastly, use the nano phytochemicals both topically on the infected skin as well as orally. Doing both is important. Real honey may also help. I hope this makes it's way to you and helps him.

I will do some research for you. I will make a new comment with ny findings.